Acute Thermal Burn
(2nd or 3rd degree)
HBOT for Acute Thermal Burn
Thermal burns cause complex tissue destruction. The injury creates a central zone of coagulation surrounded by areas of impaired circulation and inflammation. Rapid edema formation extends damage beyond the initial burn site, as blood cells aggregate and microvascular circulation becomes compromised. Ischemic tissue death quickly progresses as oxygen and nutrient supply to borderline tissues is disrupted.
For this, HBOT works through several key mechanisms:
Increases oxygen dissolution in plasma, providing critical nutrients and energy for healing.
Decreases pro-inflammatory cytokines and minimizes tissue edema, preventing progressive tissue destruction and limiting the secondary damage.
Stimulates angiogenesis, which is crucial for long-term recovery, improving nutrient delivery and supporting healing
The oxygen-rich environment significantly inhibits anaerobic bacterial growth. Reducing infection risks.